I-Avian lnfectious Bursal Disease Ag Ikhithi Yokuhlola Esheshayo | |
Isifinyezo | Ukutholwa kwe-Antigen ethile ye-Avian lnfectious Bursal Disease phakathi nemizuzu eyi-15 |
Isimiso | Isinyathelo esisodwa sokuhlolwa kwe-immunochromatographic |
Okuhloswe ngakho Ukuthola | I-Avian lnfectious Bursal Disease Antigen |
Isampula | Inkukhu Bursa |
Isikhathi sokufunda | 10 ~ 15 imizuzu |
Ubuningi | Ibhokisi elingu-1 (ikhithi) = amadivaysi ayi-10 (Ukupakishwa komuntu ngamunye) |
Okuqukethwe | Ikhithi yokuhlola, amabhodlela e-Buffer, ama-droppers alahlwayo, namaswabhu kakotini |
Isexwayiso | Sebenzisa phakathi kwemizuzu eyi-10 ngemuva kokuvula Sebenzisa inani elifanele lesampula (0.1 ml ye-dropper) Sebenzisa ngemva kwemizuzu engu-15–30 e-RT uma egcinwe ngaphansi kwezimo ezibandayo Cabangela imiphumela yokuhlolwa njengengavumelekile ngemva kwemizuzu engu-10 |
Isifo se-bursal esithathelwanayo (IBD), obeye aziwe njengoIsifo sikaGumboro,i-bursitis esithathelwanayo futhii-nephrosis yezinyoni ezithathelwanayo, yisifo esithathelwana kakhulu entsheniizinkukhu kanye nama-turkeys abangelwa i-infectious bursal disease virus (IBDV),[1] ebonakala ngokuthiimmunosuppression kanye nokufa ngokuvamile emavikini angu-3 kuya kwangu-6 ubudala.Lesi sifo satholwa okokuqala ngoGumboro, Delaware Ngo-1962. Kubaluleke kakhulu kwezomnotho embonini yezinkukhu emhlabeni wonke ngenxa yokwanda kokungenwa ezinye izifo kanye nokuphazamiseka okungekuhle kokusebenza ngempumelelo.ukugoma.Eminyakeni yamuva nje, kuye kwavela izinhlobo ezinonya kakhulu ze-IBDV (vvIBDV), ezibangela ukufa okukhulu kwenkukhu, eYurophu,i-Latin America,I-South-East Asia, Afrika kanyeEmaphakathi Mpumalanga.Ukutheleleka kungena emzileni we-oro-fecal, lapho inyoni ethintekile ikhipha amazinga aphezulu egciwane cishe amasonto amabili ngemuva kokutheleleka.Lesi sifo sisakazeka kalula sisuka ezinkukhwini ezinegciwane siye ezinkukhwini ezinempilo ngokudla, amanzi, nangokuthintana ngokomzimba.
Izifo zingavela ngokuzumayo futhi ukugula ngokuvamile kufinyelela ku-100%.Efomini elinzima izinyoni ziyagoba, ziwohloke futhi ziphelelwe amanzi emzimbeni.Zikhipha uhudo olumanzi futhi zingase zibe nentunja evuvukele.Iningi lomhlambi li-recumbent futhi linezimpaphe ezicijile.Amazinga okufa ayahluka ngobungozi bohlobo oluhilelekile, umthamo oyinselelo, ukungatheleleki kwangaphambilini, ukuba khona kwesifo esihambisana naso, kanye nekhono lomhlambi lokuthola impendulo esebenzayo yokuzivikela komzimba.I-immunosuppression yezinkukhu ezincane kakhulu, ezingaphansi kwamasonto amathathu ubudala, kungenzeka ukuthi umphumela obaluleke kakhulu futhi ungase ungabonakali ngokomtholampilo (subclinical).Ukwengeza, ukutheleleka ngezinhlobo ezinonya kancane kungase kungabonisi izimpawu zomtholampilo ezicacile, kodwa izinyoni ezine-bursal atrophy ene-fibrotic noma i-cystic follicles kanye ne-lymphocytopenia ngaphambi kwamasonto ayisithupha ubudala, zingase zibe lulaisifo esingosomathubafuthi ingase ibulawe ukutheleleka ngama-ejenti ngokuvamile ayengeke abangele izifo ezinyonini ezingakwazi ukuzivikela.
Izinkukhu ezingenwe yilesi sifo ngokuvamile ziba nalezi zimpawu ezilandelayo: ukucofa ezinye izinkukhu, umkhuhlane ophakeme, izimpaphe ezihuquzelayo, ukuvevezela nokuhamba kancane, ezitholakala zidindilize ndawonye amakhanda azo ashone phansi, isifo sohudo, indle ephuzi nenegwebu, ukungasebenzi kahle kwendle. , ukunciphisa ukudla noma i-anorexia.
Izinga lokufa libalelwa ku-20% nokufa phakathi kwezinsuku ezi-3-4.Ukuthola kabusha kwabasindile kuthatha cishe izinsuku ezingama-7-8.
Ukuba khona kwe-antibody kamama (i-antibody edluliselwa etshweleni isuka kumama) kushintsha ukuqhubekela phambili kwesifo.Izinhlobo zegciwane eziyingozi ikakhulukazi ezinamazinga aphezulu okufa zaqala ukutholwa eYurophu;lezi zinhlobo azikatholwa e-Australia.[5]